Profile

Rick Schnellmann

Pharmacology and Toxicology

Full Page

Overview

Research

Collaboration(24)

Page 1 of 5

Grants(30)

Training in Environmental Toxicology of Human Disease
Active·2024·$0 / $671.6K·
External
environmental toxicology, human disease, training, environmental health, medical education
Neuroscience Diversity to Elevate Education and Research Development
Active·2024·$0 / $269.2K·
External
neuroscience, diversity, education, research development
Therapeutic Targeting of 5HT1F Receptor-Mediated Mitochondrial Biogenesis To Address Parkinson?s Disease
Active·2022·$150K / $499.9K·
External
Co-Investigator (COI)
parkinson's disease, therapeutic targeting, 5-ht1f receptor, mitochondrial biogenesis, neurodegenerative diseases
SAVAHCS-IPA for Jaroslav Janda
Active·2022·$225.2K·
External
Principal Investigator (PI)
healthcare, medical services, health insurance, patient care, insurance network
Animal Purchasing and Husbandry Service Agreement in Support of Study Protocol #16-3117, "5-HT Stimulation of Mitochondrial Biogenesis And Acute Kidney Injury."
Active·2022·$61.2K·
External
Principal Investigator (PI)
animal research, husbandry service, mitochondrial biogenesis, acute kidney injury

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Publications(8)

Recent

5-hydroxytryptamine 1F Receptor Agonist Induces Mitochondrial Biogenesis and Promotes Recovery from Spinal Cord Injury

2020

neuroscience, spinal cord injury, receptor agonist, mitochondrial biogenesis, recovery

β-adrenergic receptor-mediated mitochondrial biogenesis improves skeletal muscle recovery following spinal cord injury

2019

skeletal muscle, spinal cord injury, β-adrenergic receptor, mitochondrial biogenesis, recovery

Author Correction: Structural and pharmacological basis for the induction of mitochondrial biogenesis by formoterol but not clenbuterol

2019

mitochondrial biogenesis, formoterol, clenbuterol, pharmacological basis, structural basis

Extracellular signal-regulated kinase 1/2 regulates NAD metabolism during acute kidney injury through microRNA-34a-mediated NAMPT expression

2019

kidney injury, nad metabolism, microrna-34a, extracellular signal-regulated kinase 1/2, regulation of nampt expression

Mitochondrial biogenesis induced by the β2-adrenergic receptor agonist formoterol accelerates podocyte recovery from glomerular injury

2019

mitochondrial biogenesis, β2-adrenergic receptor, podocyte recovery, glomerular injury, formoterol

The 5-hydroxytryptamine receptor 1F stimulates mitochondrial biogenesis and angiogenesis in endothelial cells

2019

mitochondrial biogenesis, angiogenesis, 5-hydroxytryptamine receptor 1f, endothelial cells, stimulation

Pharmacological Stimulation of Mitochondrial Biogenesis Using the Food and Drug Administration-Approved β-Adrenoreceptor Agonist Formoterol for the Treatment of Spinal Cord Injury

2019

pharmacology, mitochondrial biogenesis, spinal cord injury, drug administration, beta-adrenoreceptor agonist

Proximal Tubule Adrenergic Receptor Mediates Formoterol-Induced Recovery of Mitochondrial and Renal Function after Ischemia-Reperfusion Injury

2019

mitochondrial function, renal function, adrenergic receptor, ischemia-reperfusion injury, formoterol

People

Profile

Rick Schnellmann

Collaboration(24)

Todd Vanderah

Kirsten Limesand

Nathan Cherrington

Julie Ledford

Fayez Ghishan

Grants(30)

Training in Environmental Toxicology of Human Disease

Neuroscience Diversity to Elevate Education and Research Development

Therapeutic Targeting of 5HT1F Receptor-Mediated Mitochondrial Biogenesis To Address Parkinson?s Disease

SAVAHCS-IPA for Jaroslav Janda

Animal Purchasing and Husbandry Service Agreement in Support of Study Protocol #16-3117, "5-HT Stimulation of Mitochondrial Biogenesis And Acute Kidney Injury."

Publications(8)

5-hydroxytryptamine 1F Receptor Agonist Induces Mitochondrial Biogenesis and Promotes Recovery from Spinal Cord Injury

β-adrenergic receptor-mediated mitochondrial biogenesis improves skeletal muscle recovery following spinal cord injury

Author Correction: Structural and pharmacological basis for the induction of mitochondrial biogenesis by formoterol but not clenbuterol

Extracellular signal-regulated kinase 1/2 regulates NAD metabolism during acute kidney injury through microRNA-34a-mediated NAMPT expression

Mitochondrial biogenesis induced by the β2-adrenergic receptor agonist formoterol accelerates podocyte recovery from glomerular injury

The 5-hydroxytryptamine receptor 1F stimulates mitochondrial biogenesis and angiogenesis in endothelial cells

Pharmacological Stimulation of Mitochondrial Biogenesis Using the Food and Drug Administration-Approved β-Adrenoreceptor Agonist Formoterol for the Treatment of Spinal Cord Injury

Proximal Tubule Adrenergic Receptor Mediates Formoterol-Induced Recovery of Mitochondrial and Renal Function after Ischemia-Reperfusion Injury